TY - JOUR
T1 - Differential expression of paralog RNA binding proteins establishes a dynamic splicing program required for normal cerebral cortex development.
AU - Cesari, E
AU - Farini, D
AU - Medici, V
AU - Ehrmann, I
AU - Guerra, Marika
AU - Testa, E
AU - Naro, Chiara
AU - Geloso, Maria Concetta
AU - Pagliarini, Vittoria
AU - La, Barbera L
AU - D'Amelio, M
AU - Orsini, T
AU - SF, Vecchioli
AU - Tamagnone, Luca
AU - Fort, P
AU - Viscomi, Maria Teresa
AU - DJ, Elliott
AU - Sette, Claudio
PY - 2024
Y1 - 2024
N2 - Sam68 and SLM2 are paralog RNA binding proteins (RBPs) expressed in the cerebral cortex and display similar splicing activities. However, their relative functions during cortical development are unknown. We found that these RBPs exhibit an opposite expression pattern during development. Sam68 expression declines postnatally while SLM2 increases after birth, and this developmental pattern is reinforced by hierarchical control of Sam68 expression by SLM2. Analysis of Sam68:Slm2 double knockout (Sam68:Slm2dko) mice revealed hundreds of exons that respond to joint depletion of these proteins. Moreover, parallel analysis of single and double knockout cortices indicated that exons regulated mainly by SLM2 are characterized by a dynamic splicing pattern during development, whereas Sam68-dependent exons are spliced at relatively constant rates. Dynamic splicing of SLM2-sensitive exons is completely suppressed in the Sam68:Slm2dko developing cortex. Sam68:Slm2dko mice die perinatally with defects in neurogenesis and in neuronal differentiation, and develop a hydrocephalus, consistent with splicing alterations in genes related to these biological processes. Thus, our study reveals that developmental control of separate Sam68 and Slm2 paralog genes encoding homologous RBPs enables the orchestration of a dynamic splicing program needed for brain development and viability, while ensuring a robust redundant mechanism that supports proper cortical development.
AB - Sam68 and SLM2 are paralog RNA binding proteins (RBPs) expressed in the cerebral cortex and display similar splicing activities. However, their relative functions during cortical development are unknown. We found that these RBPs exhibit an opposite expression pattern during development. Sam68 expression declines postnatally while SLM2 increases after birth, and this developmental pattern is reinforced by hierarchical control of Sam68 expression by SLM2. Analysis of Sam68:Slm2 double knockout (Sam68:Slm2dko) mice revealed hundreds of exons that respond to joint depletion of these proteins. Moreover, parallel analysis of single and double knockout cortices indicated that exons regulated mainly by SLM2 are characterized by a dynamic splicing pattern during development, whereas Sam68-dependent exons are spliced at relatively constant rates. Dynamic splicing of SLM2-sensitive exons is completely suppressed in the Sam68:Slm2dko developing cortex. Sam68:Slm2dko mice die perinatally with defects in neurogenesis and in neuronal differentiation, and develop a hydrocephalus, consistent with splicing alterations in genes related to these biological processes. Thus, our study reveals that developmental control of separate Sam68 and Slm2 paralog genes encoding homologous RBPs enables the orchestration of a dynamic splicing program needed for brain development and viability, while ensuring a robust redundant mechanism that supports proper cortical development.
KW - SLM2
KW - SLM2
UR - https://publicatt.unicatt.it/handle/10807/263054
U2 - 10.1093/nar/gkae071
DO - 10.1093/nar/gkae071
M3 - Article
SN - 0305-1048
VL - 52
SP - 4167
EP - 4184
JO - Nucleic Acids Research
JF - Nucleic Acids Research
IS - 8
ER -